Reduced testosterone

The second theory is similar and is known as "evolutionary neuroandrogenic (ENA) theory of male aggression". [77] [78] Testosterone and other androgens have evolved to masculinize a brain in order to be competitive even to the point of risking harm to the person and others. By doing so, individuals with masculinized brains as a result of pre-natal and adult life testosterone and androgens enhance their resource acquiring abilities in order to survive, attract and copulate with mates as much as possible. [77] The masculinization of the brain is not just mediated by testosterone levels at the adult stage, but also testosterone exposure in the womb as a fetus. Higher pre-natal testosterone indicated by a low digit ratio as well as adult testosterone levels increased risk of fouls or aggression among male players in a soccer game. [79] Studies have also found higher pre-natal testosterone or lower digit ratio to be correlated with higher aggression in males. [80] [81] [82] [83] [84]

The most commonly used AAS in medicine are testosterone and its various esters (but most commonly testosterone undecanoate , testosterone enanthate , testosterone cypionate , and testosterone propionate ), [53] nandrolone esters (most commonly nandrolone decanoate and nandrolone phenylpropionate ), stanozolol , and metandienone (methandrostenolone). [1] Others also available and used commonly but to a lesser extent include methyltestosterone , oxandrolone , mesterolone , and oxymetholone , as well as drostanolone propionate , metenolone (methylandrostenolone), and fluoxymesterone . [1] Dihydrotestosterone (DHT; androstanolone, stanolone) and its esters are also notable, although they are not widely used in medicine. [54] Boldenone undecylenate and trenbolone acetate are used in veterinary medicine . [1]

If a young man's low testosterone is a problem for a couple trying to get pregnant , gonadotropin injections may be an option in some cases. These are hormones that signal the body to produce more testosterone. This may increase the sperm count. Hedges also describes implantable testosterone pellets, a relatively new form of treatment in which several pellets are placed under the skin of the buttocks, where they release testosterone over the course of about three to four months. Injections and nasal gels may be other options for some men.

The problem was that D-Aspartic acid taken alone could deplete your testosterone precursors. Just to be on the safe side, I thought I should have testosterone precursors in reserve. The three primary precursors of testosterone are progesterone, pregnenolone and DHEA. These three precursors were also listed in a scientific article as a means of reducing cholesterol if you returned them (along with testosterone) to “youthful” levels. Since my cholesterol was on the high side, it made sense to try all of them, plus I had literature supporting the idea of taking them.

Reduced testosterone

reduced testosterone

The problem was that D-Aspartic acid taken alone could deplete your testosterone precursors. Just to be on the safe side, I thought I should have testosterone precursors in reserve. The three primary precursors of testosterone are progesterone, pregnenolone and DHEA. These three precursors were also listed in a scientific article as a means of reducing cholesterol if you returned them (along with testosterone) to “youthful” levels. Since my cholesterol was on the high side, it made sense to try all of them, plus I had literature supporting the idea of taking them.

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